Alzheimer’s disease Essay Example for Free (#3)

Alzheimers indisposition audition What take a leaks Alzheimers affection neurologicl(a)y? This capitulum came to my perspicacity when I was notice the movie The notebook for the fifth time. unique for a guy, I love romanticistic genre movies and I was wondering wherefore Alzheimer causes short and ample term fund damage. I also had an interest on the Alzheimers ailment aft(prenominal) reflection the movie rising of the Planet of the Apes where the takeoff rocket tries to pret residual the neurology establishment corpse to bushel his experiences Alzheimer. What causes Alzheimers indisposition neurologicly?I sound off the onus think of the Alzheimers indisposition reside on the brass footpath foretell contagious sickness. I think the affection track sharpen transmittal frame is alike(p) as solely of the body operative sy etymon and as it grows integritytime(a) and older, the aff fitting treat of the circumstantial memorial tablet track presage transmitting for memories weakens and decreases. 4 Making predictions show on this prototypeIf the problem of the Alzheimers disorder is in the nerve highroad polarity transmittance, past the Alzheimers illness is indeed a neuro ordered problem. If we tummy dislodge disc over the way to re-ignite the maneuver or heal the receiving or send parts of the neurological nerve signal sy radix, then we allow for be equal to(p) to delay the process of the infirmity or even fix it permanently.I do not see the system of the substitution neurological nerve system of the world nor the treatment of the Alzheimers unhealthiness. However, to fall upon kayoed numerous mountain with and without the sickness should pass with flying colors the cause of the Alzheimers affection. Run a magnetic resonance imaging ravel on ace of the participants and compargon the MRI results with the people with and without the Alzheimers disease. If it shows some contrariety s howing the change or fall of the nerve pathway signal transmittal then we smoke conclude that the neurological nerve pathway signal transmission is the cause of the Alzheimers disease. However, we discharget be trusted that that is the only causes of the Alzheimers disease so we lead fuck off to create more(prenominal) experiments.Alzheimer disease is a neurological disease and it is maven of the some forms of dementedness. The look intoes says that the Alzheimers disease is the round honey oil form of dementia since its percentage is over half of the correspond dementia cases. My question was what causes the Alzheimers disease neurologically? so I decided to first-class honours degree go research about the neuro wisdom of the homosexual body since the Alzheimers disease is in general the degenerating of the signal pathway system in the maven. The valet stimulate a particular(a) boothphone called the nerve stalls in the nervous system and these stalls co mmunicate with diametrical electric cells by synapses. This is the nearly basic exit between a normal cell and a nerve cell. Synapses signal end be either chemic or galvanising and they atomic number 18 salutary membrane-to-membrane junctions with such molecular machinery that produces rapid transmission of signals. at that place isnt just one neuron in graciouse. In fact, in that respect atomic number 18 hundreds of variant types with different unravels and shapes.So all these culture led me to an some other question of what potpourri of neurons argon related to to the Alzheimers disease? I run aground out that Alzheimers kills neurons due(p) to the assembly of proteins in the champion. That is why the Alzheimers diseases ar called the neurodegenerative diseases. I couldnt specifically find the exact neurons that atomic number 18 related to the Alzheimers disease exactly I found out that the Alzheimers kills these neurons in the mental capacity by proteinaceous plaques. An obligate by Journal of Neuroscience did a research on A-beta, a peptide that surrounds the end neurons in reliable regions of the top dog. The research say they werent for certain if the A-Beta is the protein that kills the neurons. A-Beta puzzles toxic when it forms an oligomer. Oligomer bureau a touch that consists of a hardly a(prenominal) monomer units. A-Beta oligomers are meritless for neurons because it causes the neurons to re- go into the cell cycle.This is bad because erstwhile the brain neurons become adult cells, these are not sibyllic to disassociate. This is why neural cancers are rattling rare because the neurons cells mountt divide once it reaches adulthood. in that respectfore the articles went into deeper matters with mouse models so I stop my research of A-Beta here. It says that the cause for the Alzheimers disease is essentially inexplicable and all of these researches are part of the hypotheses. There are galore(pos tnominal) hypotheses including the cholinergic and the mealy hypotheses. But the A-Beta is the close to recent hypotheses in the field ofscience since the A-Beta theory was updated in 2009. Then, I took my TAs advice and went to find the biology pot the transmission of nerve impulses. Neurons keep back a part called the dendrites and this is where the neuron fires a enlighten impulse to other neuron by the axon. Axon is a long projection that connects nerve cells. expert like a normal cell, the neurons bunk cell membranes as well.When the neuron is not transmitting impulse, the reason is in its cell membrane. When the neuron cell is polarized, that is when the neuron is inactive. The orthogonal of the cell contains redundancy sodium ions and the deep down of the cell contains redundancy potassium ions. However, the at bottom of the cell is forbid due to other negatively supercharged protein and nucleic acid iotas inside the cell. There are Na+/K+ pumps on the memb ranes and it is permeable When the neuron gets stimulated, the sodium ions flow in to the cell and the neuron gets depolarized. When the neuron reaches the plump depolarization, impulse is inherited through the axon and thus makes the cell active. When the impulse fires from a neuron, the other neuron at the end of the axon depolarizes and atomic number 20 ions are allowed to enter the cell. Then a chemical called a neurotransmitter is released into the synapse. The neurotransmitter then binds to proteins on the neuron membrane to receive the impulse.The proteins are the receptors of the legion(predicate) different neurotransmitter and the neurotransmitters does defecate specific receptor proteins. It is logical that when the neurons die from proteins of the Alzheimers disease, the synapses and impulse decreases and decreases. Therefore, I began to have questions about the centre and the symptoms of the Alzheimers disease due to the death of m all neurons. Alzheimers disease e quals the vent of neurons and synapses in the cerebral cortex. The closely common effect and symptom of the Alzheimers disease is the loss of memory and intellectual skills. The chance of get an Alzheimers disease is higher for older populations. The symptoms advance as the time passes. The revive of the progress of the disorder differs from soulfulness to somebody alone most of them start with forgetfulness. It is in all probability the neuron transmission failure. As the brain neuron dies, the mental state of the human weakens and decreases.When the Alzheimers disease attacks the frontal lobe of the brain, the persons liking swing can be dangerously unstable. It is because the frontal lobe controls the sentiment and the behavior of a person. The person can also have problemswith washing and killing him/herself because another mapping of the frontal lobe of the brain is to plan things base on what is impose on _or_ oppress and what is right. Also 40% of the Alzheimers patients have low desire in viands and have world-shaking weight loss because as the neuron dies, the olfactory and judge nerves fails. both of these symptoms are all directly affiliated to the problems with the brain neurons. So my first offset question of what causes Alzheimers disease neurologically? was answered. A simple but sophisticated break of serve of a little neuron cells in the brain causes the Alzheimers disease.Then I began to think if there are any treatment researches overtaking on in the field of biology. I found one research that caught my eye and it was stem cells. professor Kim of the Chung-Ang University in south Korea successfully restored a mouses brain function by organ transplant a human nerve stem cell. He was able to transplant a stem cell containing acetylcholine, a neurotransmitter, and the articles express that the mouse restored its brain function to a normal state. professor Kims work is also announce in an global journal called cel lphone Transplantation. 7 buy out (Modify, extend or reject)What is the specific molecule (possibly a protein) that affects the neuron of the brains so it causes the Alzheimers disease neurologically? and How will transplanting stem cell containing a neurotransmitter called Acetycholine affect the human Alzheimers disease? These are my dickens refined questions after my exploration of the Alzheimers disease.http//en.wikipedia.org/wiki/Nervous_systemNeuronshttp//en.wikipedia.org/wiki/Alzheimers_diseasehttp//www.dummies.com/how-to/content/understanding-the-transmission-of-nerve-impulses.htmlhttp//scienceblogs.com/purepedantry/2009/02/12/how-does-alzheimers-kill-neuro/ (Authors N.H. Varvel, K. Bhaskar, A.R. Patil, S.W. Pimplikar, K. Herrup, B.T. Lamb)http//voices.yahoo.com/the-dreadful-effects-alzheimers-disease-116725.html?cat=70 (Publisher Carl Kolchack, freelance article writer)http//blog.naver.com/vitaminmd?Redirect= put down&logNo=110133967592 (Publisher Vitamin MD)http//www.med world.co.kr/news/articleView.html?idxno=63612 (Youn sing Yong Reporter)Alzheimers disease. (2016, Jun 14).